VULVOVAGINAL VARICOSITIES ON PREGNANT WOMAN IN CONJUNCTION WITH CONDYLOMA ACUMINATADEVI ARTAMI SUSETIATI AND BUDI SATRIA
Vulvovaginal varicosities (VV) / Vulvar varicose vein is a dilatation of vaginal veins, various data for its incidence has been reported. Prevalence is hard to be measured since its seldom to be diagnosed, often related with vein thromboembolic incidence in or out of the pregnancy period. Rare cases caused delivery disturbance. Condyloma Acuminata (CA) is a benign proliverative lesion caused by Human Papilloma Virus (HPV) transmitting by contact with epithel of the lesion (skin) or vaginal fluid that contain HPV especially from sexual activities, other than direct or non direct contact and from delivery woman to her child. Both condition could affect delivery processes. 33 years old pregnant woman (G3P1A1) 19th gestational week, visit for bumps in the vagina, diagnosed for condyloma acuminata in a STD Clinic and has been treated before. Dermato-venereology examination support for condyloma acuminata and Dupplex Ultrasound show varicose vein in vulvar area with retrograde flow, no thrombus was founded. patient was diagnosed asvulvovaginal varicosities, and crural varices on multi para pregnant woman G3P1A1 19th week gestational age in conjunction with CA. Conservative therapy was preferred for VV and CA was continued for Trichloroacetic Acid (TCA) spot therapy, and zinc supplement. VV is a condition where thereâs an abnormality dilation of vaginal blood vessel by multiple cause. VV was seldom to be diagnosed caused by multiple factor such as reluctance, atypical location, asymptomatic. Many complication could happen and cosmetic complain was main complain for this patient. Conservative management was preferred because most of its spontaneous regression soon after delivery for VV and CA. Case shows a report for multipara pregnant woman G3P1A1 19th week gestational age diagnosed with VV and crural varices in conjunction with CA. Both asymptomatic disease were detected from counseling for STD. Physical and supporting examination confirm for diagnosis. Management for CA was TCA 80% spot therapy, and zinc 20 mg OD, routine evaluation was eduvated. VV was treated conservatively, for no indication for therapy and self regression. Warning was reminded for heavier symptom or complication to reach medical services immediately, and for way of delivery.